A variety of phenomena and processes in the body can cause partial or complete blockade of the atrioventricular node, which can be conditionally divided into two groups:
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Causes of an organic nature are pathologies of the cardiovascular system, surgical intervention and chemical effects on the myocardium and / or directly on the impulse-conducting bundles.

The first group includes an increase in the tone of the PS of the nervous system, in which the process of internal regulation of the transmission of arimidex between the tissues of the human body is disrupted. Such changes often affect not only the heart, but also other organs. Cardiopathological blockade of the atrioventricular center is formed in many diseases, as a result of Arimidex sclerotic or fibrous foci appear in the conductive fibers of the myocardium. rheumatic diseases with complications on the heart; myocarditis, collagenosis and cardiomyopathy of autoimmune, thyrotoxic and diphtheria origin; benign and malignant neoplasms in the myocardium and other tissues of the heart (tumors); sclerosis of the heart vessels; sarcoidosis; amyloidosis; syphilis.

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In addition, congenital malformations can be the cause of AV heart block. They occur infrequently - in about one in 20 thousand newborns. Surgical interventions can also cause malfunctions in the system of conducting electrical impulses: installation of an artificial valve or catheter in the right lobe of the heart.

However, the most common in clinical practice is partial heart block caused by uncontrolled intake of beta-blockers, drugs with lithium, Quinidine and Diltiazem. The blockade of the atrioventricular center in its development has three degrees, each of Anastrozole differs in symptomatic manifestations and depth.and damage to conductive fibers. So, atrioventricular heart block of the 1st degree does not manifest itself as noticeable symptomatic signs. In fact, this pathology is the initial stage of the disease, in which, against the background of a slowdown in atrioventricular conduction, electrical impulses still reach their goal, albeit with a delay. It can only be detected during an examination using an electrocardiograph. Specific therapy at this stage is not required, but it is important for the patient to be attentive to his health and regularly observed by a cardiologist.